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| Research Topics: |
| INDEX |
| AI-chat 1 |
| PRIME 18 |
| aging 9 |
| body 2 |
| brain 10 |
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Scanned: 2759 Evaluated: 288 Selected: 68 |
| PRIME   POLLUTION | ||||||||
Aac03-002 | A New Study Suggests That the Hysteria Over Microplastics May Be Overblown | |||||||
| Quote: | One of the scientists challenging the study was more blunt. The brain microplastic paper is a joke, said Dr Duan Materi#263;, at the Helmholtz Centre for Environmental Research in Germany. Fat is known to make false-positives for polyethylene. The brain has approximately 60 fat. ... Theres nothing unusual about this at all. This is the sausage-making part of science. Ideas and theories are constantly questioned and challenged by other scientists; new data is added to the mix, changing minds or hardening positions until a rough consensus is achieved. | |||||||
| Comment: | The dominant nanoparticles, and I mean VASTLY dominant nanoparticles that we are exposed to on a daily basis, are silicates from rocks. These are the inorganic portions of what we call soil, dirt, etc. We are also exposed to a wide variety of other natural nanoparticles from wood, wool, cotton, etc. Is anyone ressearching the effects of those nanoparticles? I am reminded of Dr. Ames, who developed the test for mutagenicity of our modern chemicals. There was qhite an alarm that set off. He later started testing natural foods we eat, and found them far more mutagenic. of course, as those plants are battling insects trying to eat them. We call many of those chemicals flavor, and seem to do ok with them. | |||||||
| Article: | https://pjmedia.com/rick-moran/2026/01/18/a-new-study-suggests-that-the-hysteria-over-microplastics-may-be-overblown-n4948438 | |||||||
| Search Similar: | Brave Mojeek Startpage Dogpile Bing Googl Lycos Utub Yaho | |||||||
| HEART   POLLUTION | ||||||||
Mcr03-032 | Workplace Pollution and Risk of Incident Coronary Artery Disease | |||||||
| Quote: | Conclusions and Relevance Workplace pollution is confirmed as a robust, independent risk factor for incident CAD. Its association is demonstrably stronger than residential air pollution and persists despite comprehensive adjustment for genetic predisposition and traditional cardiovascular risk factors. These findings underscore the need to strengthen worker protections, integrate occupational history into clinical risk assessment, and prioritize mechanistic research into non-lipid pollution pathways. | |||||||
| Abstract: | https://www.medrxiv.org/content/10.64898/2026.01.16.26344285v1 | |||||||
| Full Text: | https://www.medrxiv.org/content/10.64898/2026.01.16.26344285v1.full-text | |||||||
| Search Similar: | Brave Mojeek Startpage Dogpile Bing Googl Lycos Utub Yaho | |||||||
| Search Authors: | Hua Gao Kai-Uwe Jarr Yoko Kojima Ting Xiong Juyong Brian Kim Paul Cheng Leonardo Trasande Annette Peters Gerard Pasterkamp Chiara Giannarelli Nicholas J. Leeper, | |||||||
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01/24/2026-02:51:25 AM
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