DIABETES KIDNEYS
Bph43-004	The RNA m6A reader YTHDF2 depletion phenocopies high glucose induced-cellular stress response in diabetic kidney disease
	Quote:	In corroboration with the above observations in animal and cell line models, independent human DKD transcriptomic datasets demonstrated conserved inflammatory and fibrotic signatures as well as reduced YTHDF2 expression in DKD glomeruli. The YTHDF2 downregulation was found to be inversely correlated with several disease-associated inflammatory and fibrotic genes. Together, these findings identify YTHDF2 dysregulation as a potential post-transcriptional mechanism associated with cellular stress and disease-associated molecular programs in DKD.
	Article:	https://www.biorxiv.org/content/10.64898/2026.10.23.761296v1
	Search Similar:	Brave Mojeek Startpage Dogpile Bing Googl Lycos Utub Yaho
	Search Authors:	Harsha Sankar SH Akhilesh Puranik Kaustuv Tripathy Nivedita Singh Anuradha Kesharwani Vipan Parihar Ruthrothaselvi Bharathavikru,

Javilk's Research Links Newsletter for Week 43
Updated 10/31/2026-02:16:20 AM
Back Issue Topic Map
What interests you? What have you found? Tell me!
Drop me a note at Javilk.com to subscribe or submit something.

Bph43-004

The RNA m6A reader YTHDF2 depletion phenocopies high glucose induced-cellular stress response in diabetic kidney disease